Sports Medicine FOR THE PRIMARY CARE PROVIDER - Hershey Medical Center
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WINTER 2016 Sports Medicine F O R T H E P R I M A R Y C A R E P R O V I D E R Concussions and Chronic Traumatic Encephalopathy BY MATTHEW SILVIS, M.D. The recent movie “Concussion” has created quite a stir nationally about chronic traumatic encephalopathy (CTE). This is written to help fellow providers understand what is known, and just as importantly, unknown about this condition. In 10 to 15 percent of athletes with concussion, symptoms last longer than 10 days and can persist for weeks, months, or years after injury. Post-concussion syndrome is ill-defined, poorly understood and currently explained as persistent symptoms or signs of concussion that persist for weeks or months after concussion. ‘Time’ is the primary treatment for post-concussion Dear Health Care Provider, syndrome, which is frequently frustrating for patients and providers, alike. A multidisciplinary My name is Matthew Silvis. I am medical approach is recommended, including providers with experience in caring director of Penn State Primary Care Sports for sports-related concussions and may also include pharmacologic Medicine. I have enclosed the winter edition management, physical, vestibular, speech and occupational of our Primary Care Sports Medicine therapies, neuropsychological evaluation and treatment, visio-ocular Newsletter, a biannual newsletter of seasonal evaluation and treatment, and behavioral health, amongst others. sports topics. We hope you find the information In recent years, there has been increasing media attention to useful and appreciate any feedback you have sports-related concussion, specifically CTE. CTE is a progressive to enhance our efforts. We have selected a neurodegenerative tauopathy associated with repetitive brain variety of topics for this issue. injury. It was originally diagnosed in boxers If you’d like to receive this newsletter by more than 85 years ago. Research groups email, please forward your address to my have proposed that head injury, including administrative assistant, Sandy Miland at both concussive and subconcussive blows, smiland@hmc.psu.edu. Please send any leads to neuropathologic changes and future topic ideas to Sandy or me at the development of alterations in mood, cognition and behavioral functioning. msilvis@hmc.psu.edu. CTE can occur in as few as one blow to the head and appears to develop from eight to Enjoy, 20 years following retirement from contact sports. The diagnosis is made at autopsy and is separate from permanent post-concussion Matthew Silvis, M.D. syndrome. Initially, mood and behavior ASSOCIATE PROFESSOR changes predominate with later clinical PENN STATE HERSHEY FAMILY AND COMMUNITY MEDICINE presentations involving cognitive and PENN STATE HERSHEY ORTHOPAEDICS AND REHABILITATION PENN STATE MILTON S. HERSHEY MEDICAL CENTER CONT’D ON PAGE 2
Skin Infections in Wrestlers BY BY JESSICA BUTTS, M.D. Wrestling season is upon us. Due to the nature of the sport, with brief, here is a listing with a few reminders: near constant skin-to-skin contact, skin infections are a common • Tinea corporis requires a minimum of 72 hours of topical antifungal; issue encountered when caring for competitive wrestlers of all ages. Fungal infections (e.g., ringworm), viral infections (typically herpes • Tinea capitis requires 10 days of oral antifungals; gladiatorum caused by HSV-I) and bacterial infections (impetigo • Herpes gladiatorum requires treatment with an oral antiviral: 10 days and MRSA) are all common offenders. As these infections are highly for initial outbreak and 120 hours for recurrence; all lesions must be transmissible among these athletes, providers need to be vigilant with crusted over with no new lesions in the preceding 48 hours. patients who wrestle and present with skin complaints. Many skin infections, specifically those listed above, would disqualify the athlete • Bacterial skin infections need at least three days of oral antibiotics until they have been properly treated. As these infections directly affect with all lesions scabbed. their eligibility to participate, wrestlers may go to extreme measures All of this information, and more, can be found at the bottom of the to hide the nature of their lesions. Applying bleach, using sandpaper PIAA wrestling skin form. and excoriating (or picking) are all possible ways to hide or disguise As we care for these athletes, being aware of how common these disqualifying skin conditions. Therefore, providers who treat these skin infections are in this population, as well as being aware of the athletes need to have a very high level of suspicion for these conditions, potentially atypical appearance of some of these lesions, is important. as well as a low threshold to treat empirically if any of these conditions Being familiar with the PIAA are suspected. guidelines for return-to-play Completion of the PIAA “skin form” is universally required for all and having a low threshold to wrestlers with any potentially contagious skin lesion to be allowed to treat keeps as many athletes in participate. This form is infinitely useful to keep in any primary care the game (or in this case on office setting where a “wrestler with rash” may present for evaluation. the mat) as possible. This form identifies diagnosis, number and location of lesions, and type and date of treatment initiated. This form is required at pre- participation skin checks by any athlete who has had a skin evaluation and treatment. Completion of this form by the provider at the time of visit streamlines this process for the athlete, athletic trainer and medical staff covering these events. The PIAA form also outlines standard required treatment for common skin infections to be eligible to return to play. These guidelines are extraordinarily useful as they outline the minimum required treatment for some of the most commonly encountered skin conditions. No athletes with new lesions in the preceding 48 hours, or with lesions that are oozing, draining or moist, will be permitted to participate. In CONCUSSION FROM COVER motor impairment. by other groups of neuropathologists. recent study demonstrated no increased Interestingly, there is a greater incidence risk of neurodegenerative disease in a However, CTE is poorly understood of abnormal tau protein deposition in long-term follow-up study of high school at this time. Athletes with significant the brains of opiate abusers compared to football players versus a control group neurologic symptoms do not always have controls according to research studies. Of (band members). While providers and the histopathologic changes of CTE, and note, more than half of the players in the public are appropriately concerned, much the presence of histopathologic changes National Football League (NFL) have been is unknown about CTE and a definitive of CTE is not always associated with reported to use opiates during their NFL causal link to American football has yet neurologic symptoms. Tau protein may be careers with most reporting abuse, clouding to be determined. Future prospective, found in individuals undergoing normal the etiology of CTE. NFL players are less longitudinal, population-based studies aging, independent of head trauma, likely to die from suicide than the general are needed to better understand CTE. For although some leading researchers report population and there is no definitive link now, following standard-of-care in the a qualitatively different deposition of between CTE and suicide at this time. In management of athletes with concussion abnormal tau protein in CTE versus other fact, retired NFL players live longer than remains the best approach. neurodegenerative diseases. However, their age-matched peers. Additionally, one this finding has not been cross-validated 2
When is a meniscal tear not a meniscal tear? When it is a root avulsion! BY ROBERT A. GALLO, MD Meniscus tears come in all shapes and sizes. There are vertical tears, horizontal tears, radial, and even complex tears which can include any combination of these tear orientations. These conditions are managed largely by the degree of symptomatology, tear orientation and location. More recently, meniscal root avulsions (tears), a subset of meniscal tears, have received increased attention in the orthopaedic community. A meniscal root avulsion involves any disruption of the tibial attachment sites of the menisci. While each meniscus has attachments circumferentially to the knee capsule, the root insertions are located at 1 2 both the anterior and posterior ends of each meniscus. The meniscal root attachments anchor each meniscus to the tibia and are essential to the normal function of the meniscus as “shock absorbers.” With a detachment of its root attachment, the meniscus loses the ability to resist hoop stresses vital to its normal function. Therefore, the downward force of the femoral condyles on the tibia causes the meniscus to eventually extrude beyond the confines of the tibio- femoral articulation (figure 1). Rapidly progressive osteoarthritis can follow if the meniscus is unable to efficiently transmit load to the tibia. While a meniscal root avulsion can occur in younger individuals in a trauma setting, the majority occur in middle-aged adults. Most 3A 3B patients recall incidents when they felt a “pop” and immediate FIGURE 1: Extrusion of the meniscus beyond the border of the femoro-tibial pain in the posterior medial or lateral aspect of the knee. Similar to articulation (line) occurs with prolonged weight-bearing following disruption of the other meniscal tears, patients describe worsening pain with deep root attachment. knee flexion; however, the pain is more posterior than meniscal FIGURE 2: Meniscal root tears are readily visualized as increased signal between the tears involving the body and posterior horn of the meniscus. On meniscus and tibial (arrow) on T2-coronal MRI sequences. examination, the hallmark physical examination finding is tenderness FIGURE 3: Meniscus root tears can be surgically repaired by passing sutures into the to palpation of the joint line at the posterior aspect of the knee. meniscus, sending the sutures down a tunnel through the tibia, and tying the sutures over a button on the anterior tibial cortex. Imaging should begin with anteroposterior weightbearing, lateral and Merchant radiographic views of the affected knee. If moderate or severe osteoarthritis is identified on radiographs, treatment The post-operative limitations are fairly extensive and involve a period should manage the symptoms of osteoarthritis. In cases of relatively of non-weight-bearing and immobilization. preserved joint spaces, MRI should be considered if a posterior root Despite the theoretical advantages of surgical repair, long-term clinical tear is suspected. While they can usually be visualized on any image data assessing healing, meniscal extrusion and the ability to slow the orientation, meniscal root avulsions are most readily seen on coronal progression of arthritis are lacking. Therefore, repair should only be images as an area of signal intensity between the posterior meniscus considered after careful evaluation, which includes assessment of the and tibia (figure 2). amount of pre-existing osteoarthritis, level of disability and symptoms The treatment for meniscal root avulsions has evolved over recent and body habitus (i.e., obese patients place increased stress on any years. Because of recent biomechanical studies confirming the benefits repair and are more likely to fail the repair procedure). Intra-articular of restoring the meniscal root attachment, repair of these avulsion-type steroid injections and physical therapy are useful alternative treatment tears has become increasingly popular. Surgical repair involves the modalities. following process: In conclusion, root avulsions are a unique category of meniscal tears • passing a high-strength suture through the torn edge of the that must be considered a different entity than other meniscal tears. meniscus; Presenting with posterior knee pain, meniscal root tears can render the meniscus nonfunctional, resulting in increased joint reactive forces • passing those sutures through a small tunnel beginning at the and leading to rapidly progressive osteoarthritis. Consultation to an normal root insertion site and exiting along the anteromedial tibia orthopaedic surgeon should be considered in those with an MRI- adjacent to the tibial tubercle; then diagnosed meniscal root tear and relatively well-preserved joint space, • tying the sutures over a button on the anteromedial cortex of the as seen on anteroposterior weight-bearing radiographs. tibia (figure 3). 3
Helmets – A False Sense of Security? BY JAYSON LOEFFERT, D.O. , PRIMARY CARE SPORTS MEDICINE FELLOW However, there are types of head injuries that helmets have been shown to prevent, including skull fractures and head lacerations. Helmets can compress during impact, allowing some deceleration and decreasing the direct force applied to the head. Unfortunately, this does not translate to prevention of all types of head injuries, namely traumatic brain injury and concussion. The question then is why haven’t helmets had more of an impact on preventing head injury? There are several hypotheses: First, it has been widely demonstrated that helmet use does not eliminate the risk of concussion. While helmets can absorb some impact during a collision, they do not significantly reduce the acceleration or deceleration and rotational forces resulting in concussion. Additionally, helmets are not being tested at speeds consistent with all usage environments. The average skier travels at a speed of 24.6 to The colder weather brings with it the anticipation of snow, and, of 31.3 miles per hour, yet linear impact tests are conducted in the lab at course, skiing and snowboarding! Over the last decade, the concern only 14.3 miles per hour. Going above the average speed could result for injury and prevention has grown, and simultaneously, public in as much as a quadruple increase in subjected impact energy above helmet use has skyrocketed: 6 to 25 percent in 2003 to 70 to 90 what the helmet is designed to protect. Therefore, helmets may not percent in 2013. The purpose of this article is to educate fellow be designed to handle the forces they are subjected to in real-world providers on the limitations of helmet use for preventing head injury, environments. Finally, the idea of increased risk-taking behavior due as well as to warn against false security and over-assumption that to helmet use has been discussed. While the idea that helmets overtly helmets prevent head injury. cause recklessness has been refuted, their use could have an indirect Groups all over the world have recognized the growing trend to wear effect. If athletes believe they are more protected because they are helmets while skiing or snowboarding, and have studied the effect this wearing a helmet, they may travel at faster speeds or engage in riskier has had on head injury. Although studies are limited, there does not maneuvers than they otherwise would without the helmet. appear to be a strong correlation between helmet use and decreased Fortunately head injuries in snow sports are rare, but still a concern head injury. In fact, one study has shown that in spite of increased for all skiers and snowboarders. When considering helmet use during helmet use, the frequency of head injuries also increased over the snow sports, it is important to realize that helmets will not prevent all same time period. head injuries. Practicing safe activities and wiser decision-making on the mountain may be a better way to prevent head injury! PRIMARY CARE SPORTS MEDICINE Shawn Phillips, M.D. Robert Gallo, M.D. sphillips6@hmc.psu.edu rgallo@hmc.psu.edu Matthew Silvis, M.D. Assistant Professor, Departments of Family and Community Assistant Professor, Penn State Hershey Orthopaedics msilvis@hmc.psu.edu Medicine and Orthopaedics Penn State Hershey Bone and Joint Institute, 717-531-5638 Associate Professor, Departments of Family and Community Penn State Hershey Medical Group—Mt. Joy, 717-653-2900 Medicine and Orthopaedics Scott Lynch, M.D. Penn State Hershey Bone and Joint Institute, 717-531-5638 Medical Director, Primary Care Sports Medicine slynch@hmc.psu.edu Penn State Hershey Medical Group—Palmyra, 717-838-6305 Rory Tucker, M.D. Associate Professor, Director of Sports Medicine Service Penn State Hershey Bone and Joint Institute, 717-531-5638 jtucker@hmc.psu.edu Practice Site Clinical Director of Adult Bone and Joint Institute Assistant Professor, Departments of Family and Community Medicine Associate Director of Orthopaedic Residency Education, 717-531-5638 Jessica Butts, M.D. and Orthopaedics Penn State Hershey Bone and Joint Institute, 717-531-5638 Jbutts@hmc.psu.edu Penn State Hershey Medical Group—Camp Hill, 717-691-1212 Assistant Professor, Departments of Family and Community Medicine Penn State Hershey Bone and Joint Institute, 717-531-5638 SPORTS MEDICINE PHYSICAL THERAPY and Orthopaedics Penn State Hershey Medical Group—Nyes Road, 717-214-6545 Andrew Wren, D.O. Robert Kelly, PT, ATC Penn State Hershey Bone and Joint Institute, 717-531-5638 awren@hmc.psu.edu Physical Therapist, Certified Athletic Trainer Associate Professor, Department of Family and Community Medicine Team Physical Therapist, Hershey Bears Hockey Club Bret Jacobs, D.O. Medical Director, Penn State Hershey Medical Group— bjacobs@hmc.psu.edu Scott Deihl, ATC, PTA Elizabethtown, 717-361-0666 Assistant Professor, Departments of Family and Community Medicine Physical Therapist Assistant, Certified Athletic Trainer and Orthopaedics ORTHOPAEDIC SPORTS MEDICINE Tanya Deihl, ATC, PTA Penn State Hershey Medical Group—Middletown, 717-948-5180 Physical Therapist Assistant, Certified Athletic Trainer, Penn State Hershey Bone and Joint Institute, 717-531-5638 Kevin Black, M.D. Athletic Trainer, Annville Cleona High School kblack@hmc.psu.edu Cayce Onks, D.O. Professor and C. McCollister Evarts Chair John Wawrzyniak, MS, ATC, PT, CSCS conks@hmc.psu.edu Penn State Hershey Orthopaedics Physical Therapist, Certified Athletic Trainer Assistant Professor, Departments of Family and Community Medicine Penn State Hershey Bone and Joint Institute, 717- 531-5638 Strength & Conditioning Specialist, Hershey Bears Hockey Club and Orthopaedics Penn State Hershey Medical Group—Palmyra, 717-838-6305 Aman Dhawan, M.D. Penn State Hershey Bone and Joint Institute, 717-531-5638 adhawan@hmc.psu.edu Assistant Professor, Department of Orthopaedics Penn State Hershey Bone and Joint Institute, 717-531-5638 4 FCM-9228-16 022216
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